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Clinicians can frame these outcomes as supportive and adjunctive rather than curative, emphasizing that GHK-Cu enhances the results of a comprehensive regimen rather than replacing cornerstone interventions such as sun protection, neuromodulators, or energy-based devices

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Another drug, activated protein C, reduces BBB breakdown and leakage in stroke model rodents and has anti-inflammatory effects, neuroprotective effects, and blocks hemorrhage after brain ischemia [126, 127]

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Additionally, the increased MDA levels observed upon SYNCRIP depletion were significantly reduced following SYNCRIP restoration (Figure 3E-G), further linking SYNCRIP function to suppression of ferroptosis